15-Great-Stories-That-Have-Nothing-To-Do-With-Politics.Html A-H1N1 Swine Flu Likely Originated in US Lab

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A-H1N1 Swine Flu Likely Originated in US Lab

When the first reported cases of A/H1N1 swine flu emerged in April 2009, it was widely believed that the new influenza strain had originated in Mexico. In late June, when evidence failed to confirm a Mexican origin, a new theory suggested that A/H1N1 originated in Asia and was inadvertently transported to North America by a suspected human carrier. This, however, is probably not the case. Instead it is likely, though not confirmed, that A/H1N1, a genetically engineered creation that originated in the United States, specifically in a lab in Madison, Wisconsin, accidentally escaped through some sort of contamination.

The evidence for this scenario is compelling:

1. Prior to the A/H1N1 outbreak, The Institute for Molecular Virology (IMV) at Buck Labs (run by the University of Wisconsin-Madison) was involved in an infectivity study for vaccine production. The research involved reverse genetic engineering of a tissue sample taken from a deceased Intuit woman who succumbed to the Spanish influenza that killed 50 million people in the 1918-19 pandemic.

2. The current A/H1N1 variant is a “highly unusual virus” never seen before that combines genetic material from North American human, avian, and swine flu and Eurasian swine flu.[1] According to The Associated Press, such combinations in pigs, birds or humans are unprecedented, and impossible to occur naturally. When no close relatives of the current strain exist and the IMV’s mission – conducting virology research and training at a molecular level – is taken into account, creation through artificial genetic engineering offers the best explanation.

3. Retired Australian researcher Adrian Gibbs, who pioneered the development of Tamiflu®, a highly effective anti-flu drug, theorized on May 12, 2009 that the new strain of A/H1N1 may have escaped a laboratory setting because it is the vaccine. The study “demonstrates the characteristics of undergoing accelerated evolution, such as what happens when a flu virus tries to adapt to egg growth”.[2] Although the World Health Organization (WHO) quickly dismissed Mr Gibbs’ theory a day later, it is incredible that enough research could be done in just 24 hours to confirm a conclusion.

4. When the presence of A/H1N1 was firmly established in the United States by May 10, 2009, Wisconsin and Illinois accounted for about a third of the country’s cases. Since then Wisconsin has consistently led the nation with a population of 5,627,967 based on the July 2008 estimate compared to larger states – California, Texas, New York, Illinois and even Michigan with a July 2008 population of 36,755,666, 24,97,420, 24,29,421, 24,440. 29,43,203. By June 12, 2009, when the outbreak was over, Wisconsin and Illinois still accounted for more than a quarter of the cases in the United States. It makes little sense to talk about this disproportionate caseload in terms of demographics. However, when Madison, WI is viewed as the focal point, the two-state caseload provides inconsistent evidence of virus initiation. Located in La Gloria where the first case of A/H1N1 is believed to have occurred, a subsidiary of American-based Smithfield Foods), other parts of the United States and eventually the rest of the world.

Centers for Disease Control (CDC) Bulletin:

5/10/2009: Wisconsin: 357 cases (14.1% of national caseload); Illinois: 466 cases (18.4% of national caseload)

6/12/2009: Wisconsin: 3008 cases (16.8% of national caseload); Illinois: 1983 cases (11.1% of national caseload)

5. To date the A/H1N1 2009 swine flu pandemic variant has not been found locally in pig stocks worldwide, discounting the theory of natural mutation and initial pig-to-human transmission. Furthermore, none of Wisconsin’s pig stocks have tested positive for the novel A/H1N1 that currently plagues the world.

6. Statements and actions indicate prior knowledge. As early as April 25, 2009, when the new A/H1N1 strain was officially identified in only 3 states (11 cases), a top CDC official, Dr. Ann Schuchat said, “We don’t think we can stop the spread of this virus.” By April 28, 2009, Vice President Joseph Biden had “ruled out isolating Mexico, citing limited benefits due to the swine flu virus.” [had] “has already entered many states” (64 cases in 5 states). When the A/H1N1 outbreak in Mexico was reported on April 23, 2009, an immediate quarantine was not implemented because the CDC and top US government officials had already been alerted. Unconfirmed and unreported infections. A quarantine made little sense because cases in the United States were developing rapidly and because the move would likely raise suspicion when such cases were subsequently confirmed and reported.

7. Samples of the new A/H1N1 virus were already present at CDC before the Mexican sample was received. In an interview conducted by CDC virologist Ruben Donis for Science Direct (published April 29, 2009) — the CDC completed sequencing of the novel A/H1N1 strain two weeks ago, or by April 15, 2009 — three days before Mexican officials sent the swab samples for testing. Headquartered in Atlanta.

Based on the above compelling data, conclusive evidence exists that the A/H1N1 swine flu outbreak that led to WHO’s first pandemic declaration in 41 years was artificially created and likely traced back to IMV’s lab in Madison, WI. can As a result, A/H1N1 is a moderate risk based on its characteristics and a potential threat especially to a generation that has never experienced a pandemic and has pre-existing medical conditions (asthma and other respiratory disorders, diabetes, heart problems, immune deficiency disorders, and pregnancy, to name a few) whose immune systems are unprepared or ill-equipped to recognize and deal with novel strains, respectively, must be taken seriously. At the time of writing this article, this is not being done (as the New York City Department of Health stated on its website as late as June 25, 2009 – “most cases of influenza-like illness do not require testing for H1N1” even though seasonal influenza has disappeared for the summer, suspected Failure to facilitate isolation in emergency room cases etc.). Continued failure to do so could result in between 1 million (current WHO estimates that up to a third of the world’s population may be infected, based on a 25% mortality rate) to 25 million or more deaths as many more people are treated. between illnesses (after developing severe complications) and/or if the virus transforms into a more virulent form resulting in the 1+% mortality rate that is already being demonstrated in Argentina, a country that has entered the winter season.


[1] Donald G. McNeill, Jr In the new theory, swine flu started in Asia, not Mexico. The New York Times. 24 June 2009.

[2] K, flu experts are claiming that swine flu was developed in a lab. CBCNews.ca. 12 May 2009. 24 June 2009. www.cbc.ca/health/story/2009/05/12/swine-flu-evolution.html

additional sources

Wayne Madsen. Hybrid A/H1N1 flu linked to genetic trigger for larger, mutated version. Online Journal. 24 June 2009. 24 June 2009. onlinejournal.com/artman/publish/article_4837.shtml

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